Biological Functions and Molecular Mechanisms of Antibiotic Tigecycline in the Treatment of Cancers
Identifieur interne : 000A02 ( Main/Exploration ); précédent : 000A01; suivant : 000A03Biological Functions and Molecular Mechanisms of Antibiotic Tigecycline in the Treatment of Cancers
Auteurs : Zhen Dong [République populaire de Chine] ; Muhammad Nadeem Abbas [République populaire de Chine] ; Saima Kausar [République populaire de Chine] ; Jie Yang [République populaire de Chine] ; Lin Li [République populaire de Chine] ; Li Tan [République populaire de Chine] ; Hongjuan Cui [République populaire de Chine]Source :
- International Journal of Molecular Sciences [ 1422-0067 ] ; 2019.
Abstract
As an FDA-approved drug, glycylcycline tigecycline has been used to treat complicated microbial infections. However, recent studies in multiple hematologic and malignant solid tumors reveal that tigecycline treatment induces cell cycle arrest, apoptosis, autophagy and oxidative stress. In addition, tigecycline also inhibits mitochondrial oxidative phosphorylation, cell proliferation, migration, invasion and angiogenesis. Importantly, combinations of tigecycline with chemotherapeutic or targeted drugs such as venetoclax, doxorubicin, vincristine, paclitaxel, cisplatin, and imatinib, have shown to be promising strategies for cancer treatment. Mechanism of action studies reveal that tigecycline leads to the inhibition of mitochondrial translation possibly through interacting with mitochondrial ribosome. Meanwhile, this drug also interferes with several other cell pathways/targets including MYC, HIFs, PI3K/AKT or AMPK-mediated mTOR, cytoplasmic p21 CIP1/Waf1, and Wnt/β-catenin signaling. These evidences indicate that antibiotic tigecycline is a promising drug for cancer treatment alone or in combination with other anticancer drugs. This review summarizes the biological function of tigecycline in the treatment of tumors and comprehensively discusses its mode of action.
Url:
DOI: 10.3390/ijms20143577
PubMed: 31336613
PubMed Central: 6678986
Affiliations:
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</affiliation>
<affiliation wicri:level="1"><nlm:aff id="af4-ijms-20-03577">Chongqing Engineering and Technology Research Center for Silk Biomaterials and Regenerative Medicine, Southwest University, Beibei, Chongqing 400716, China</nlm:aff>
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<author><name sortKey="Cui, Hongjuan" sort="Cui, Hongjuan" uniqKey="Cui H" first="Hongjuan" last="Cui">Hongjuan Cui</name>
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<country xml:lang="fr">République populaire de Chine</country>
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</affiliation>
<affiliation wicri:level="1"><nlm:aff id="af2-ijms-20-03577">Cancer Center, Medical Research Institute, Southwest University, Beibei, Chongqing 400716, China</nlm:aff>
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<wicri:regionArea>Cancer Center, Medical Research Institute, Southwest University, Beibei, Chongqing 400716</wicri:regionArea>
<wicri:noRegion>Chongqing 400716</wicri:noRegion>
</affiliation>
<affiliation wicri:level="1"><nlm:aff id="af3-ijms-20-03577">Engineering Research Center for Cancer Biomedical and Translational Medicine, Southwest University, Beibei, Chongqing 400716, China</nlm:aff>
<country xml:lang="fr">République populaire de Chine</country>
<wicri:regionArea>Engineering Research Center for Cancer Biomedical and Translational Medicine, Southwest University, Beibei, Chongqing 400716</wicri:regionArea>
<wicri:noRegion>Chongqing 400716</wicri:noRegion>
</affiliation>
<affiliation wicri:level="1"><nlm:aff id="af4-ijms-20-03577">Chongqing Engineering and Technology Research Center for Silk Biomaterials and Regenerative Medicine, Southwest University, Beibei, Chongqing 400716, China</nlm:aff>
<country xml:lang="fr">République populaire de Chine</country>
<wicri:regionArea>Chongqing Engineering and Technology Research Center for Silk Biomaterials and Regenerative Medicine, Southwest University, Beibei, Chongqing 400716</wicri:regionArea>
<wicri:noRegion>Chongqing 400716</wicri:noRegion>
</affiliation>
</author>
</analytic>
<series><title level="j">International Journal of Molecular Sciences</title>
<idno type="eISSN">1422-0067</idno>
<imprint><date when="2019">2019</date>
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<front><div type="abstract" xml:lang="en"><p>As an FDA-approved drug, glycylcycline tigecycline has been used to treat complicated microbial infections. However, recent studies in multiple hematologic and malignant solid tumors reveal that tigecycline treatment induces cell cycle arrest, apoptosis, autophagy and oxidative stress. In addition, tigecycline also inhibits mitochondrial oxidative phosphorylation, cell proliferation, migration, invasion and angiogenesis. Importantly, combinations of tigecycline with chemotherapeutic or targeted drugs such as venetoclax, doxorubicin, vincristine, paclitaxel, cisplatin, and imatinib, have shown to be promising strategies for cancer treatment. Mechanism of action studies reveal that tigecycline leads to the inhibition of mitochondrial translation possibly through interacting with mitochondrial ribosome. Meanwhile, this drug also interferes with several other cell pathways/targets including MYC, HIFs, PI3K/AKT or AMPK-mediated mTOR, cytoplasmic p21 <sup>CIP1/Waf1</sup>
, and Wnt/β-catenin signaling. These evidences indicate that antibiotic tigecycline is a promising drug for cancer treatment alone or in combination with other anticancer drugs. This review summarizes the biological function of tigecycline in the treatment of tumors and comprehensively discusses its mode of action.</p>
</div>
</front>
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</listBibl>
</div1>
</back>
</TEI>
<affiliations><list><country><li>République populaire de Chine</li>
</country>
</list>
<tree><country name="République populaire de Chine"><noRegion><name sortKey="Dong, Zhen" sort="Dong, Zhen" uniqKey="Dong Z" first="Zhen" last="Dong">Zhen Dong</name>
</noRegion>
<name sortKey="Abbas, Muhammad Nadeem" sort="Abbas, Muhammad Nadeem" uniqKey="Abbas M" first="Muhammad Nadeem" last="Abbas">Muhammad Nadeem Abbas</name>
<name sortKey="Abbas, Muhammad Nadeem" sort="Abbas, Muhammad Nadeem" uniqKey="Abbas M" first="Muhammad Nadeem" last="Abbas">Muhammad Nadeem Abbas</name>
<name sortKey="Abbas, Muhammad Nadeem" sort="Abbas, Muhammad Nadeem" uniqKey="Abbas M" first="Muhammad Nadeem" last="Abbas">Muhammad Nadeem Abbas</name>
<name sortKey="Abbas, Muhammad Nadeem" sort="Abbas, Muhammad Nadeem" uniqKey="Abbas M" first="Muhammad Nadeem" last="Abbas">Muhammad Nadeem Abbas</name>
<name sortKey="Cui, Hongjuan" sort="Cui, Hongjuan" uniqKey="Cui H" first="Hongjuan" last="Cui">Hongjuan Cui</name>
<name sortKey="Cui, Hongjuan" sort="Cui, Hongjuan" uniqKey="Cui H" first="Hongjuan" last="Cui">Hongjuan Cui</name>
<name sortKey="Cui, Hongjuan" sort="Cui, Hongjuan" uniqKey="Cui H" first="Hongjuan" last="Cui">Hongjuan Cui</name>
<name sortKey="Cui, Hongjuan" sort="Cui, Hongjuan" uniqKey="Cui H" first="Hongjuan" last="Cui">Hongjuan Cui</name>
<name sortKey="Dong, Zhen" sort="Dong, Zhen" uniqKey="Dong Z" first="Zhen" last="Dong">Zhen Dong</name>
<name sortKey="Dong, Zhen" sort="Dong, Zhen" uniqKey="Dong Z" first="Zhen" last="Dong">Zhen Dong</name>
<name sortKey="Dong, Zhen" sort="Dong, Zhen" uniqKey="Dong Z" first="Zhen" last="Dong">Zhen Dong</name>
<name sortKey="Kausar, Saima" sort="Kausar, Saima" uniqKey="Kausar S" first="Saima" last="Kausar">Saima Kausar</name>
<name sortKey="Kausar, Saima" sort="Kausar, Saima" uniqKey="Kausar S" first="Saima" last="Kausar">Saima Kausar</name>
<name sortKey="Kausar, Saima" sort="Kausar, Saima" uniqKey="Kausar S" first="Saima" last="Kausar">Saima Kausar</name>
<name sortKey="Kausar, Saima" sort="Kausar, Saima" uniqKey="Kausar S" first="Saima" last="Kausar">Saima Kausar</name>
<name sortKey="Li, Lin" sort="Li, Lin" uniqKey="Li L" first="Lin" last="Li">Lin Li</name>
<name sortKey="Li, Lin" sort="Li, Lin" uniqKey="Li L" first="Lin" last="Li">Lin Li</name>
<name sortKey="Li, Lin" sort="Li, Lin" uniqKey="Li L" first="Lin" last="Li">Lin Li</name>
<name sortKey="Li, Lin" sort="Li, Lin" uniqKey="Li L" first="Lin" last="Li">Lin Li</name>
<name sortKey="Tan, Li" sort="Tan, Li" uniqKey="Tan L" first="Li" last="Tan">Li Tan</name>
<name sortKey="Tan, Li" sort="Tan, Li" uniqKey="Tan L" first="Li" last="Tan">Li Tan</name>
<name sortKey="Tan, Li" sort="Tan, Li" uniqKey="Tan L" first="Li" last="Tan">Li Tan</name>
<name sortKey="Tan, Li" sort="Tan, Li" uniqKey="Tan L" first="Li" last="Tan">Li Tan</name>
<name sortKey="Yang, Jie" sort="Yang, Jie" uniqKey="Yang J" first="Jie" last="Yang">Jie Yang</name>
<name sortKey="Yang, Jie" sort="Yang, Jie" uniqKey="Yang J" first="Jie" last="Yang">Jie Yang</name>
<name sortKey="Yang, Jie" sort="Yang, Jie" uniqKey="Yang J" first="Jie" last="Yang">Jie Yang</name>
<name sortKey="Yang, Jie" sort="Yang, Jie" uniqKey="Yang J" first="Jie" last="Yang">Jie Yang</name>
</country>
</tree>
</affiliations>
</record>
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